In Moderation
Providing health, nutrition and fitness advice in moderate amounts to help you live your best life.
Rob: Co-host of the podcast "In Moderation" and fitness enthusiast. Rob has a background in exercise science and is passionate about helping others achieve their health and fitness goals. He brings a wealth of knowledge and expertise to the show, providing valuable insights on topics such as calories, metabolism, and weight loss.
Liam: Co-host of the podcast "In Moderation" and new father. Liam has a background in nutrition and is dedicated to promoting a balanced and sustainable approach to health and wellness. With his witty and sarcastic style, Liam adds a unique flavor to the show, making it both informative and entertaining.
In Moderation
Bridging Medicine, Fitness, and Cholesterol Myths with Dr. Alo: A Dive into Diets, Supplements, and Health Hacks
Discover how a cardiologist and certified personal trainer, Dr. Alo, ingeniously bridges the worlds of medicine and fitness. Get an exclusive peek into Dr. Alo's journey, from our early TikTok days to his revolutionary cookbook designed to meet specific caloric and protein needs. Learn the ins and outs of Mediterranean and DASH diets, and enjoy a light-hearted discussion on the curious nutritional value of bull testicles. Dr. Alo also shares his contributions to sports coaching and insights from his published works on flag football and soccer.
Ever wondered about the truth behind cholesterol myths and dietary supplements? Listen as we dissect the controversial discussions around cholesterol, debunking myths and addressing irony, especially regarding figures like Paul Saladino. Engage in an enlightening conversation about Big Pharma conspiracy theories, the role of statins, and the nuances of seed oils and saturated fats. Dr. Alo emphasizes the importance of critical thinking and personal responsibility in navigating health information.
Genetics, cholesterol levels, and dietary guidelines are put under the microscope in this episode. Understand the complex interplay between genetics and health conditions like diabetes and familial hypercholesterolemia, and how lifestyle changes can make a difference. Learn to interpret cholesterol and lipid panel results, including the significance of Lp(a) levels and effective cholesterol reduction strategies. From practical dietary tips to the latest guidelines, we cover it all with a mix of scientific rigor and humor, even touching on the absurdities of social media criticism and the idea of an OnlyFans for cholesterol education.
You can find Dr. Alo
https://www.instagram.com/dr.alo/?hl=en
https://www.tiktok.com/@drmohammedalo
You can find us on social media here:
Rob Tiktok
Rob Instagram
Liam Tiktok
Liam Instagram
All right, everybody, since Rob introduced the last one, as he just told me he did, because I don't remember anything, just like the number of episodes we've done, episode 69, again For the sixth time. For the sixth time, episode 69. For today, on today's episode of In Moderation, we have Dr Allo. I'm excited, Dr Allo. I was friends with you on TikTok early on when I started making content. How are you doing?
Speaker 2:I'm awesome. I remember we started off at about around the same time and it was kind of interesting. We'd go live a lot. We'd bring on Dr Terry Simpson. Yeah, it was fun. We had a good time.
Speaker 1:Yeah, I remember we did start about the same time. That's right. I remember you were's right. I remember you were like I remember you were doing like um, um, a doctor recommends and you just like recommend different channels, and I was like one of them or something. I was like, oh yeah, that's me awesome and it was one of my.
Speaker 2:I had a thing called doctors to follow because there was so much nonsense on social media, right that's what it? Was a bunch of idiots. So one of the things I was doing, people like who do you recommend we should follow? So I was like, well, I'll just start a playlist called Doctors to Follow, and you were one of the first ones. I was like, obviously he's not a doctor, but I'm giving an honorary medical degree.
Speaker 1:Honorary doctorate. Honorary doctorate, that's all I've ever wanted. I don't want all the work of getting a information is accurate. As long as I get the title, that's all that really matters, right? Uh, yeah, I remember you did a bunch of other people like lane norton and you know, even like ben carpenter and stuff like that that was great yeah, it's good company. Yeah, it's good company. Well, doctor, what so? What are you a doctor of for people who don't know? What are you specializing?
Speaker 2:I'm a cardiologist. Obviously all cardiologists are internists too. So you do you internal medicine first, so you're really good at taking care of adults. You know all forms of adult disease and then you specialize in heart disease. Um, I'm also a certified personal trainer. So a lot of people come at me and they're like you don't know anything about nutrition and exercise. I'm like, actually I have a degree in exercise science. It's not like a PhD, but it's you know. A certified personal trainer is not an easy thing to get, but it's you know, it's an extra degree. I'm super fascinated with fitness. I coach a lot of sports. I train my friends, I train myself. In working out I was doing like some bodybuilding got down to like 7% body fat at one point. I still work out pretty much every day, sort of like you guys do. I love coaching sports. I love mentoring young people. You almost become like a second father to a lot of the kids you coach and it's a lot of fun. I've coached almost every sport you can name, mainly football, flag football.
Speaker 3:Canadian or American?
Speaker 2:American. I also coach football, the soccer version of it. I actually have two books one on how to dominate soccer and one on how to dominate flag football.
Speaker 1:Oh, I didn't know you had that. I know you have your other book, your Heart to Heart, right? Is that what it is?
Speaker 2:I got a cookbook on how to eat heart healthy. But this is super unique and I don't think like the chapters are divided up into calories needed to lose weight. So like, let's say, liam, you need to lose weight. You go to the 1800 calorie chapter. There's multiple different numbers. There's 2,400, 2,200, 2,000, 1,800, all the way down to 1,200. You go to 1,800, you make breakfast, lunch and dinner and dessert and you will lose weight and each chapter contains enough protein for that person of that weight. Let's say you weigh like 200 pounds. You need to lose some. You get about 160, 180 grams of protein a day but there's no saturated fat, there's no salt. It's heart healthy. You know a lot of it is like protein. That is not going to give you a lot, put you over your calories or, you know, put you too high on saturated fat. So it's super, super unique. There's no other book like it.
Speaker 1:Yeah, so since you do nutrition and you're like a cardiologist as well, like where, what do you kind of recommend in terms of, like the dietary, your dietary recommendations, since you kind of do both of them? You know this crossover.
Speaker 2:So, generally speaking, you want to follow a more Mediterranean or DASH style diet. These are like more wholesome foods. You know the usual stuff that somebody would eat. If you know, minus most of the ultra-processed stuff, you can eat it. I'm not super strict about that. Uh, you know plants, fruits, vegetables, legumes, lean meats, salmon, with their studies you know, with fish seeds, all that kind of stuff Exactly.
Speaker 2:You guys probably follow very similar diets. Now, if somebody is like I want to eat a pub tart, like okay, dude. Like well, I want to eat ice cream every night, have at it. Fit it into your calorie count. You know, as long as your cholesterol and lipid markers are all okay which they can be you'll be totally fine. So I'm not like an extremist or a fanatic when it comes to being really strict about food. People are like, well, do you ever eat red meat? Sure, you know, not every day. I'm not having ribeyes, you know it's like usually the tops are loins or fillets, but it's super rare.
Speaker 3:Where do we?
Speaker 1:fall on bull testicles. Are bull testicles lean Are?
Speaker 2:those full of saturated fat? I honestly don't know the answer to this. If you ask my good friend, paul Saladino, we had that great debate one time and I had literally just got. This is the funniest story. I obviously do a lot of videos on Paul Saladino, right, because you guys do too, because we debunk a bunch of nonsense. I literally just got done calling this guy a naked caveman, telling people to eat raw bull liver testicles, whatever. I made up some crazy thing that doesn't even exist raw liver, bull testicle extract or whatever and he messages me the next day has nothing to do. He never saw any of my videos. He has no idea I make fun of him. I was obscured.
Speaker 3:He probably had no idea who the hell.
Speaker 2:I am and he's like.
Speaker 2:Hey, he's like you're a cardiologist, do you mind hopping on my podcast and talking about cholesterol? Oh no, he goes different ideas about cholesterol. So I was like, oh God, this guy does I mean, did he not watch what I just posted? So I was like this guy has like billions of followers. I'm pretty sure he didn't see any of the stuff I posted, because there was like a YouTube video that I'd done about him recently. That was like this guy's just probably the worst influencer you can have. So I went to my YouTube studio real quick and like turn, turn that off because like if he did some research about me.
Speaker 2:So much hate though I'm sure he just doesn't even pay attention to any of it because, like it's just from everywhere yeah I mean I tune it out now at this point, I don't even. I mean, I read the comments and try to respond to people who are respectful and normal, but if people are like you're ugly your eyebrows look fake. You know why?
Speaker 1:does your hair?
Speaker 2:look like that. I'm like exactly I'm sure you guys get the same thing with the braided beard and the hair and all that. But it's like dude if you can't get past the way someone looks and just talk about the actual content or the data or the stuff.
Speaker 1:Right, it's a beauty contest.
Speaker 3:My most recent Paul Saladino video. I of course had that guy that came in and was like hey, paul looks healthier than you who do you want to look like?
Speaker 2:I'm like, first of all I can deadlift probably three paul saladinos right, I mean like literally I deadlift, I squat, I bench press.
Speaker 1:Maybe not three of them, but definitely two I mean, I'm just saying the guy goes outside with beef tallow on and sits in the sun all day Like I don't know. I'm a little concerned about that.
Speaker 2:Sometimes I wonder if that stuff is. So. Him and I had a really long conversation before we went on air and it was the same program we're using now, because his Wi-Fi was like glitchy they keep going in and out and you have to start over. So we were talking a lot while he's trying to set up and do stuff in and out and you have to start over. So we were talking a lot while he's trying to set up and do stuff.
Speaker 2:I have it all recorded because I didn't think he was going to actually post it Right, so I had my own like background screen share grab you know OBS video, whatever that program is called to record it on my end, in case the guy was like, eh, I'm not going to post this, you know this. This didn't work out as well as I thought it would, but credit to him he did post the entire thing completely unedited, with his liver testicle supplement commercials in between, um, but he did and I give him credit. I mean we had a good, lively debate. Um, depends on who you asked or go you, he won. You won Like I don't know it wasn't a win or anything, it was like a debate about. You know, here's the science and data, and here's liver testicles.
Speaker 3:And now you have last month. He goes on More Plates, more Dates podcast and talks about how he's moving away from carnivore because it's not healthy.
Speaker 2:Not only that, but his testosterone went down. He just feels like garbage. He hated it and he doesn't think anybody should do it. That was the final sentence. He's like I don't think anybody should do keto, which like okay, dude, we've been telling you this for five years, but he did write a book on it.
Speaker 1:First, all about the carnivore diet yeah, he made a lot of money.
Speaker 2:Liver King says they sold $100 million dollars worth of supplements. So you know it's hard to. And then the funniest thing, okay. So when I was preparing for my talk with Paul Saladino I'll give you guys another backstory I went back and listened to all his podcasts that he did with other quote unquote doctors on cholesterol. He interviewed that guy, asim Malhotra, the nut job from UK that you know. They pretty much suspended him and kicked him out of the the, the England. Um, because he's like pro vaccine, then he's anti-vaccine, then he's for statins, then he's anti-statins like literally every 10 minutes.
Speaker 2:Wherever the money is, he changes his mind. Um, so forget that guy. So he didn't. In the end, I see Malhotra did say and Paul did Paul said in that interview that statins do seem to work right. I just wanted to make sure that we're kind of on the same page, at least on like he actually does read data and you can't really deny it, right. So he did say that. I'm like okay, we can have a discussion that seems fruitful because he's not denying the data. He said at the end every single study about statin medications has shown benefits. I was like, okay, this dude is not insane, we can have a conversation. But I went back further to an interview I think it was 2018 with a guy called Malcolm Kendrick, another one of these crazy cholesterol deniers. Funny clip in there Paul Saladino says yeah, yeah, yeah, those guys are idiots. They're just trying to sell you supplements.
Speaker 1:And I'm like, I'm like you know what I went?
Speaker 2:I was going to go back and clip that and be like, hmm, he said he doesn't believe somebody. They were talking about somebody. Him and Malcolm Kendrick were talking about somebody and I forget they both didn't like this somebody. And he's like, yeah, they just say that to sell supplements, they just sell supplements.
Speaker 1:Meanwhile he does all his videos with the supplements in the background, just in plain view.
Speaker 2:And they're always just right around. Yeah, this was probably before he started selling supplements. So I want to go back and probably like stitch that video, but I had to screen record the other one because it was on youtube. It's a long video on youtube. I highly recommend you guys find that you can easily go now because it has the auto text, auto chapters and that auto transcript. If you just search it for supplement, you probably find the exact moment in time when he said that and you could probably make a very funny video. I know you guys love doing that too it is.
Speaker 1:It's ridiculous. I always see that that thing you know you talk about stands obviously in your videos because you know our medicine doctor. But I always see like, oh, but you know, big pharma is just trying to get your money. And it's just like statins like aren't they like generic? And like super cheap now, like they cost, like pennies, like who's making all this money off them? What's going on?
Speaker 2:A one month supply literally costs probably two pennies. I mean it probably. Your insurance charges you three bucks for a 90 day supply or whatever it might be, maybe 10, depending on your copay and what insurance plan you signed up for. But generally speaking, first of all, nobody, and I can say nobody, but you don't get money for prescribing a medication At the end of the month. There's no Pfizer calls me up and they're, like you, only prescribed 10.
Speaker 1:You did pretty well this month, doc. You only prescribed 10.
Speaker 2:The press stores were only sending you 10 bucks. Like no, what are you guys talking about? It's like, why do people think this? Like there's so many weird conspiracy theories out there, it's almost laughable. And the other big one is like who funded that study? Like you're just demonstrating that you've never read research in your life. Literally right at the top it says funding and right under that conflict of interest, like you've never even opened a study your life. If you're asking who funded that study, like you're just an idiot. I'm not going to say that to people, but it's like it's very intellectually lazy. I try to be nice.
Speaker 1:I'm like you could actually just read the study Right along with me, with it's banned in other countries. Who funded this and it's banned in other countries are kind of like you know, right alongside each other for me, because it's very easy for people to just say that and then move on Okay, who funded that? Or it's banned somewhere else, and then like I don't have to do anything else, like it just makes it. It's a very simple term, right?
Speaker 2:It's also simple thinking and I think part of it and I'm not making fun of people, obviously I love people and I want to help them, but part of it and you guys do too, Obviously a lot of it is people want, I think, to believe that there's some other force out there that's preventing them from becoming the healthiest they can be, or why, society is so overweight and obese they almost want to blame, but not like in a bad way.
Speaker 2:But it's easier for them to accept the fact that we're not healthy as a country, whether it's Canada, where Rob is, or the United States, because there's this magical power called big pharma or big government, or big food or big agriculture or big whatever. And I have sympathy for these people. Obviously, I'm not this cold, evil person. I love people and I would love for them to get better and accept some facts and accept some personal responsibility. I did not make you fat, I did not give you hypertension or diabetes.
Speaker 3:I'm sure you were there shoving the cookies down their throat.
Speaker 1:No, it's the seed oils. That's what did it.
Speaker 3:Just get the funnel poured in.
Speaker 2:The seed oil people are totally another breed, like I've posted on Twitter and TikTok and every app you can imagine. Cite one single study, one, just give me one where it showed seed oils caused harm to human beings Not rats, not test tubes, not vehicles, not oceans.
Speaker 1:No, it's all mechanisms. They're like this does this, which could do that in humans.
Speaker 2:It's all this, but then when you force feed seed oil to people, they live longer and they have less heart attacks, less strokes. They lose more fat.
Speaker 1:They actually improve all their cardio metabolic risk, and that's just because you know they're poly and monounsaturated fats which we see, and that's why there's a lot of people who say like, oh, saturated fat, it's been demonized. You should be eating butter every day and and it's just like I've gone over this. You've talked about it a bunch, I've talked about it a bunch, but basically I guess we'll just kind of give the TLDR on this. With saturated fat, when you get to about over what 9%, 10% of your calories from saturated fat, you start to see some negative consequences from this. But a lot of these studies will look at the difference between 10% and 16%, or 12% and 18%, and you don't see that much difference in there. But if you go from 6% to 12%, there's a big difference there.
Speaker 2:It's a huge difference. It's a sigmoidal curve. So below about 7%, you're not going to see a difference. If you took somebody who eats about 6% or 7% total calories or total energy from saturated fat and drops it to 2% or 3% or almost 0%, no difference in all-cause mortality, cardiovascular mortality, anything. You do the same thing with above about 12%, 12% to, let's say, 100%.
Speaker 2:If you eat all saturated, you eat only butter, all day, all night, like these carnivore people. You eat only saturated fat, like these carnivore people, you eat only saturated fat. You're going to have a flat line of you know very high though cardiovascular mortality, all cause mortality cardiovascular events, strokes, heart attacks, all of that. There's an inflection point right around eight-ish, nine-ish percent. When you drop from like a 10 to a seven, huge difference. There's like the hazard ratio drops from almost two down, or like that one and a half to two, like so 50 to almost you know 100 percent worsening of outcomes to almost like a 20 to 30 percent reduction in almost all outcomes. So the country of finland, you guys can, can look this up look up the finland study or the north korelia finland study.
Speaker 2:In the 1970s they had the highest mortality 700 people out of every 100,000 would die, which was the highest in the world, and these were like people. These are in the 1970s. They had the highest mortality 700 people out of every 100 000 would die, which was the highest in the world. And these were like people. These are in the rural areas of you know, north northern finland, north korea, other areas. These were like rural farmers. They ate tons of butter, like literally 18 kilograms per person per year.
Speaker 2:That's like I don't know rob understands kilograms, I don't, but in america that's like 45 pounds maybe 45 pounds for our American friends and 18 kilograms, that's a lot of butter. So what they did is they sent out nurses and these public officials to go out there and educate people on what can we do? They reduced saturated fat intake. They actually obese. They actually gained weight. Smoking stayed dipped a little, stayed about the same, but in women it went up.
Speaker 2:Hypertension went down not a lot, but they got them down under control. They had an 84% reduction in mortality and they got down to about 100 people out of every 100,000 over the next like 30 years, and that study is still going on till today. By 2011, they had an 84% reduction in mortality. 73% of it was attributed to the reduction in saturated fat. Now, after the first couple of years when they noticed the numbers starting to go down, they made it all of Finland, not just North Karelia, and from that point on pretty much all of Finland. They lagged a little, but it was an 82% reduction for the rest of Finland. That's like a huge chapter in my new cholesterol book. Looking at that. About 20% of the reduction in mortality had to do with blood pressure and like other factors, but this is as obesity rates have gone up. So even here in the United States and Canada we've had a significant decline in cardiovascular death rates. Right In the 1960s pretty much everybody died of heart disease. 90% of people would die of heart disease.
Speaker 2:Now it's about 30, 33%, and since the 1960s we've had about an 80% 90% drop in death from heart disease, while people have gotten more overweight.
Speaker 3:While they're eating more CETOS. It always boggles my mind that people try to say that our cardiovascular rate has gone up and it's like no it's gone down.
Speaker 2:Despite worse metabolic chronic disease. More people are hypertensive, more people are overweight, more people have become obese, slightly more diabetics not a huge change, but some. But still, despite a higher burden of worse chronic disease, less and less people are dying every year of actual heart disease. So we're actually doing a really good job, but they're like oh, it's the seed oils, brother, what I find interesting, though, is they always say we're not treating the root cause.
Speaker 1:That's like Eastern medicine treats the root cause. We don't treat the root cause in that, but it's like but that's okay. Well then, how has heart?
Speaker 1:disease decreased in, you know, the rate of heart disease decreased, while all these other things are increasing. Like we understand that having high cholesterol over the long term, like I know you pointed it out. I've looked it up. It's really interesting studies of people like World War Two, people like World War II, like there was people who came back like 19-year-olds, 20-years-olds and they checked their arteries and they actually had plaque, like the beginnings of plaque in their arteries at like 20 years old, depending on how high their cholesterol was.
Speaker 2:Right, it was commensurate with their cholesterol levels. Even moms, in addition to that, newborns who were born to moms like miscarriages not born, I guess, technically not technically born, but miscarriages or accidental abortions, and even probably planned abortions maybe. But they've looked at babies that were born to mothers of very high cholesterols, like 300s and et cetera. They had fatty streaks in their arteries, their aortas, because it's bigger, you can't get into their coronaries, they're tiny.
Speaker 2:But they had super high. The higher the mom mom's cholesterol, the fattier, more fatty streaks they had. They've. We've known this since 1913. The very first study that was done on this was in rabbits. Really andrew vich or andrew vich, I forget his name he gave, he fed rabbits saturated fat, basically because rabbits don't eat meat, so they're like can't handle the saturated fat right like.
Speaker 2:Well, not just that, but they fed them like rabbit chow that was full of egg yolks and, you know, saturated fat, and their cholesterols went up and within. And then, not only that, they even infused ldl, you know lipoproteins or cholesterol. They didn't have ldl at the time, but they infused cholesterol into rabbit arteries and within hours they had fatty streaks in their arteries wow just infusing cholesterol.
Speaker 2:So these, these people are like what is the root cause? I think the root cause people are also. I'm very sympathetic to them because they're also like in this weird conspiracy world of no one wants to treat root cause Like okay, so somebody has a cold and they get a bacterial infection. We treat the bacteria which is the root cause with antibiotics. You can't tell me that, like eating more celery in a day would have fixed that. Because they're all like I think they hate patients, not in a bad way, but they want to blame the patient. They have this blame game where it's like it's your fault, you're freaking fat, you're lazy, you don't eat right, you're unhealthy, it's your fault.
Speaker 1:That's why you have heart disease they do that because they want to be able to be in control of, like of everything about. So they're saying, if something happens to someone else, it's always, and this is always throughout human history. If you look at it, if somebody got sick, it's because they're Jewishian, oh, it's because they're white, oh it's because whatever, and so, like, it's just easier to do that and say, like I'm gonna be healthy because I do these things.
Speaker 2:You want to be in control, when in reality you're just like well, actually, let's, you know, check your cholesterol, let's do all these things to actually reduce your risk I think it's easier for people to accept it because, like, imagine we, we tell you, uh, so-and-so's, oh, she was so fat and overweight and had diabetes, she had so many diseases. It's like easier to be like, okay, that's all right then. But if it's like you know, but you get what I'm saying Like I think mentally it's easier for people to kind of blame the person in a way for making bad choices, and I'm not saying all of these things are not due to bad choices, yes, right.
Speaker 2:Let's say, you have the genes for diabetes, becoming overweight and less active and all of that stuff absolutely makes it worse and doing the opposite absolutely makes it better. But not every but not everyone has those genetics. So let you know. They want to blame something, though they almost want to throw the blame onto something that's out of their control, because it's like easier to sleep at night, or they don't want to believe in this genetic thing. Like look, there are kids born with cholesterols of 300. It's a genetic disorder. They will die before they're 13 if we don't do something about it. Like, what do you want me to do? Just be like maybe you should stop eating fat. I don't eat fat, I eat celery.
Speaker 3:Put fat. I don't eat fat I eat celery.
Speaker 2:Put some cayenne pepper on your tongue and inject some urine.
Speaker 1:The kind of people are something else oh yeah, no, those kind of people are something, um, but you know what I see and it's it. I feel for these people like I had a patient recently who was like he's very fit, um, you know he was doing everything. He's like I want to lower my cholesterol, I don't want to take a stat, I don't want to do it, I don't want to take medication, um, and I was he's like I want to lower my cholesterol, I don't want to take a statin, I don't want to do it, I don't want to take medication. And I was like he's like what can I do? I'm like, well, you do this. And he's like I do all that. I eat oatmeal every day, I have high fiber, I have no saturated fat in my diet, exercise, lifestyle. Eventually, if you just genetically got fucked that's kind of the way it is you kind of just have to go. All right, I might need a medication for this and obviously talk with your doctor, but there's only so much you can do.
Speaker 2:Yeah, definitely. There are some people that did not win the genetic lottery and unfortunately, some of them are children, the kids with the homozygous familial hypercholesterolemia. They come out with 300, 400, 500 cholesterol. I mean they will never see age 13 or 14 without medication.
Speaker 1:Medication yeah.
Speaker 2:There's nothing you can do diet-wise, health-wise. You can't run it off, you can't eat it off. There's nothing you can do with a cholesterol that high that's going to get it down to a range that's not going to kill you before your 13th birthday.
Speaker 1:And that's why you talk about like the people, like a lot of the people, like in the blue zones, or I talk about where you know their diet is generally quite healthy, but also there's other things, like they have the. What is the genetic mutation that?
Speaker 2:makes they have a lot. There's at least 10 different genetic mutations. The most common one is the PCSK9.
Speaker 1:Yes, that's one I was thinking of.
Speaker 2:There's also the FOXO3 mutation. There's a whole bunch. I have a whole chapter on that as well, but there's a lot of them. I should send you guys a preview copy, a PDF. But there's definitely lots of genetic mutations within those regions because they're usually isolated regions like Sardinia, Italy, Loma Linda, usually isolated regions like sardinia, italy, loma, linda, california, the island of okinawa, um, I forget the other one.
Speaker 2:There's like costa rica. Costa rica like a little peninsula off of costa rica and nagoya maybe or something like that, but they're all like isolated areas where there's a lot of genetic lack of divergence or you know differences, and they must have a lot of these and and there there have been studies published and I have all of them listed and live linked in the PDF of the book. But there's a lot of genetics to these people that have longevity genes. So I think that's one thing.
Speaker 1:And these genetic mutations. A lot of them lower their cholesterol right, so their LDL is very low.
Speaker 2:Absolutely, absolutely, plus other things like you know, maybe telomere function, maybe other stuff but a lot of them do cause very low levels of LDL, like hypobeta lipoproteinemia. It's a huge word but it just means you have really low LDL, basically Really low ApoB, hypobeta lipoproteinemia, low beta lipoproteins in your bloodstream, but they have really low LDL and they have no atherosclerosis. As long as they don't do anything else, dumb, they'll probably live a very long time.
Speaker 1:So I think we've talked enough to get rid of the antiseed oil people. That's perfect. So now we probably have the rest of the foot. What's left is people like, hey, okay, what do I do, what's my thing? And I would love to just give people a quick little on cholesterol, right? Okay, so you've got give us a little. There's total cholesterol, there's HDL, there's LDL. What do all these things mean?
Speaker 2:Right. So when we take your blood out of your body and put it in a test tube, we spin the test tube right and it stratifies by density. The HDL is the highest density. It's at the bottom. The LDL is a little less than intermediate and then very low density than you know whatever. So what we do is we take out those fractions. The first one we take out is the HDL. We measure the cholesterol in those lipoproteins. So this is like a lipoprotein. It's kind of what they look like, this structural blue thing is called a ApoB.
Speaker 3:HDLs have one called the ApoA1. For those listening, he's holding up what looked like a baseball, an orange baseball.
Speaker 2:With blue strings. Basically the stitching on it is just this blue string. Imagine the stitching is a blue wrapper kind of On the inside of it, and I'll try to describe it as well. There's this yellow stuff and there's this turquoise stuff. The yellow stuff is cholesterol and the turquoise is triglycerides. Now, some of them can have more triglycerides than others, but the cholesterol is on the inside.
Speaker 2:So when we take out these lipoproteins, we take it out of the test tube, we spin it down, we take the cholesterol out of them and we check that amount of cholesterol in that layer of the test tube. So HDL comes out first. What's left over is all the non-HDL right, and non-HDL is a very good surrogate for ApoB, because everything else basically has a B lipoprotein on it or a structural protein, I should say. So when you take out the HDL, everything left over is non-HDL. That is probably the closest approximation or surrogate for ApoB, if you didn't get an ApoB, because the rest of it depends on calculations. The rest of it, like.
Speaker 2:You check the total cholesterol of what's left over, that's called non-HDL cholesterol. You take out triglycerides, you know the turquoise stuff on the inside, you divide that by five, subtract out, you know the cholesterol that's left and that's how you end up with calculated LDL. It is calculated at extremes. It's not as accurate below 70. It's not not super accurate. And if your triglycerides are above about 150 200, it's not super accurate either, but it's close enough. So we try to get people to check an apob or just follow your non-htl. That's super accurate. Well, because what I've always heard in.
Speaker 1:Like you know, during my schooling was like hdl was the good cholesterol, but I feel like in recent years that's kind of gotten some pushback. I've've seen it from you and I've gotten messages from people how do I increase my HDL? And from my understanding you don't really need to worry about that so much. What's going on there?
Speaker 2:Right, you don't need to worry about HDL at all. We call it now highly disappointing. I like it. It doesn't seem to matter in terms of outcomes at all. Okay, generally speaking and then I'm going to say this generally because they both do the same thing generally speaking, we, we think, and and it's true, the hdl takes the cholesterol out of your arteries.
Speaker 1:Generally speaking, the ldl puts it back into your arteries so you would think you would want a more hdl right the problem is you're measuring.
Speaker 2:when you measure HDL cholesterol, you're measuring the amount of cholesterol that's in an HDL particle which, if it's high, means it's not doing its job, it's not clearing it. So the job of an HDL is to take it back to liver to get rid of it. But if it's still in your bloodstream, that means it's not taking it back to liver to clear it. So it's high. So it means it's dysfunctional.
Speaker 1:So some people are like my HDL is 120.
Speaker 2:What should I do? I'm like that's probably not a good thing either, because it's high. It means it's not really working. It's not clearing the cholesterol very well. Do we do anything about it? Not really.
Speaker 2:But the other point I want to make is we generally don't call LDL bad. I mean, when I talk to patients I do. But also the problem is most HDLs will meet up with LDLs in circulation. They kind of like connect together and have sex almost. They connect with this tube, like literally connect with a tube, and they like dock with each other I guess is the word and the HDLs hand over cholesterol to the LDLs to help them take it back to the liver. So for a large portion of their life the LDLs which are quote unquote bad, are actually doing a lot of good. They're helping the HDLs get rid of cholesterol from your arteries in circulation and take it back to liver to be eliminated. And we don't want to just call them, label them, good and bad. The cholesterol inside is not either good nor bad. It's just a molecule when it's in your arteries.
Speaker 2:it's bad, but when it's in a lipoprotein. It's being transported around either in a direction we want or a direction we don't want.
Speaker 1:So that's kind of a but we don't want to deposit it in our artery walls. That's when you get clots, that's when bad shit happens.
Speaker 2:Exactly Too much of this will. There's nowhere to go. It starts depositing in your arteries of your wall. But some people have really high cholesterol. You see it deposit like in the skin under their eyelids. You see it deposit on the tendons, like in their elbows, on the back of their knees, on the front of their knees.
Speaker 1:I didn't know that.
Speaker 2:Patellar tendons or Achilles tendons. It's called familial hypercholesterolemia. I'm sure you could look up the pictures. They're horrific. Sometimes you see, like this, patches of this white cholesterol, almost like oozing out of your eyelids. It's terrible when it's that high. That means there's nowhere else for it left to go. It's already plugged up. You know all your arteries and now it's coming out of like the really thin skin, like you know, near your eyes, under your eyelids and above your, in your eyelids on top below your eyes.
Speaker 1:So for general, like just kind of recommendation here. You know, I don't know if rob, if you have yours, I have my like a lipid panel that I got a little while ago. You already did a video on this like a year ago. I haven't done a new one. I want to do a new one. Haven't done it yet, but I'm just going to give you these numbers then you can kind of tell me so that people can kind of get a reference and we can talk about like what is sort of a normal range. So like I got my whole like total cholesterol. Do you even look at total cholesterol? Is that even important?
Speaker 2:We don't, it doesn't matter, but you generally want that under 150.
Speaker 1:Okay, minus 134.
Speaker 2:What is it?
Speaker 1:134 for total cholesterol was one.
Speaker 2:Yeah, so that's fine. We don't have a target to treat, we don't chase it. We don't have a target to treat, we don't chase it, we don't really care about it, we just kind of it's just there to help us calculate non-HDL. And my HDL was 52, which, as we've learned, is meaningless.
Speaker 1:It's meaningless yeah.
Speaker 2:It's a number we generally ignore, except at extremes. If it's like 10, it's like, hmm, maybe you got cancer, right, right, okay, that makes sense, but my, okay, so this one. I have my ApoB, but first I'll give you my calculated LDL, which is 71. Yeah, that's fantastic. So normal people, we want them under a hundred. If you got no risk factors, never had a heart attack, never had a stroke, we generally want you under a hundred. If you have a risk factor, like, let's say, you have diabetes or you have hypertension, used to be a smoker or current smoker, whatever, we generally want you under 70. If you've never had a cardiac event, once you had a cardiac event, whether it's a stent, open heart surgery, stroke, whatever, then we want you under 55 55?
Speaker 2:Yeah, if you ever have a repeat cardiac event. We generally want those people under 40.
Speaker 1:Wow Okay. So the numbers to remember are 100, 70, 55, and 40. 40 interesting, okay, I did. I knew under 100 was considered normal and I know there's talks about like even that being maybe a little high, like 90s being a little high it probably is.
Speaker 2:I mean I would. Mine is 41 and I'm okay with it. I'm happy, okay I've had no heart attacks, no strokes, none of that. The main reason I want it that low, so that I never have one um, I have mine up.
Speaker 1:My problem is that, oh, yours is in canadian numbers.
Speaker 2:Yeah, don't worry I know I can convert it. It's super easy. Just tell me what's your uh. Go for it, I got you what?
Speaker 3:what are we on we're?
Speaker 2:on ldl hdl whatever you want to tell me the numbers generally in those studies are always in international.
Speaker 3:My LDL is 1.5.
Speaker 2:1.2, 1.5. That's fantastic 1.5. That's fine so for people who don't, that's like 57. For people who don't know, you multiply and or divide by 38.6. So that's 57. That's fantastic.
Speaker 3:Definitely.
Speaker 2:Are you on meds or not? That's fantastic if that's untreated.
Speaker 3:That would be untreated.
Speaker 2:That's amazing. That's real good.
Speaker 1:Okay, well, I want to say I got my ApoB checked. I want to say that that is a little bit lower. It's 63. How's that?
Speaker 2:That's great. Those two generally track together. That's called concordance when they track together. So that's real good. The ApoB is slightly more accurate and tracks better with the disease and risk and outcome. So 63 below 70 is considered amazing on the ApoB. Let me give you, my I think that's below the fifth percentile, to be honest with you. Okay so 55 is the fifth percentile cutoff point. So you're super close. So you're probably around seventh percentile, I'll just leave it. Maybe even less sixth, I'll just leave it alone. That's fantastic.
Speaker 3:Okay, my APOB is 0.65.
Speaker 2:0.65. 0.65. Let me do a quick calculation. That's real low 25, brother, that's amazing 25, yeah. Yeah, that's real good. That's below the fifth percentile. You're in the second percentile, two percent.
Speaker 1:Don't have.
Speaker 2:Tooth percentile.
Speaker 1:He might have to worry about being a Canadian, but he doesn't have to worry about heart attacks is basically what he's saying.
Speaker 2:Yeah, you have actually hypobeta lipoproteinemia. If you don't do anything dumb, you probably live to 100.
Speaker 2:I mean we don't have the people are like, well, you don't know that, you don't have the data to prove that. Well, look, first of all, we've never had ways of checking this. People are like, well, you keep saying people with a pcs k9 mutation and hypo beta lipoprene are gonna live to 100. We don't have data. Okay, look, but we didn't have ways of checking this before.
Speaker 2:Right now that we know all this and we have the genetic testing and we know who has this, because people who are over 100 now were born in 1900, 1900, 1901, 1920, whatever it might be, they didn't have this testing back then. We can do it now and they've gone to these places and checked some of these people. But people with your kind of levels, rob, most generally speaking, like I said I'm not going to say absolutely, because you know who the hell knows you could have liver cancer, I don't know. But people generally speaking, with that low of ApoB, almost impossible to have atherosclerosis and will probably live a very, very long life, assuming you continue to work out, eat healthy, do all that kind of stuff, don't smoke, don't drink, whatever.
Speaker 3:Well, it's a good thing that Paul Saladino looks healthier than me.
Speaker 2:Yeah, he looks. Look at his arteries. His LDL is like 200.
Speaker 1:Is it 200? Which?
Speaker 2:in. Canadian that's like 4.5, which is like insanely high.
Speaker 1:Oh, okay, oh, but I one thing, another test I learned because I got it done. You only apparently needed it once. I learned it from Dr Lippitt. What's his name, what's his actual?
Speaker 2:name Thomas Dayspring.
Speaker 1:Dayspring. I was just blanking on the name.
Speaker 2:He's actually the author.
Speaker 1:The second author on my book.
Speaker 2:Oh, he's like the co-editor I didn't know he's amazing.
Speaker 1:Yeah, no, he's very. I've listened to a few of his podcasts that he's gone on with other people. But he said to try and get your LP little a checked like protein a, because that's like a genetic mutation and like just if you either have it or you don't. And mine was came back as 23, which it says under 75 is Milligrams or millimoles, nanomoles or millimoles. Nanomoles.
Speaker 2:Oh, that's pretty low. So normally under 125 is normal for that.
Speaker 1:Okay.
Speaker 2:You're fantastic, you're good.
Speaker 1:Okay, so, and that's just something you check like once in your lifetime just to make sure you don't have it Mainly to and to have first-degree relatives check.
Speaker 2:If you had it, we got to check all your kids, brothers sisters siblings, parents, all that kind of stuff.
Speaker 3:Interesting. Well, that's something I don't have a number for.
Speaker 2:Yeah. Yeah, you could definitely check it, rob, because your LDL and ApoB could be fantastic, but if your lipoprotein A is super elevated, it's not good. I'll show you what that looks like. So here's a normal lipoprotein and for those of you who are not on youtube, we will post it. But let's say, you see a baseball in in my hand that's orange. Um, this is not what they look like in real life. They're probably not orange. They're probably not orange at all. They're probably like an off-white or, you know, maybe, um, but this is a normal ldl particle the ones that have this kingle on it, and I just put like an aluminum strip around it for demonstration purposes. But if they have this little cringle around it, those are lipoprotein A's.
Speaker 2:They're heavier and more dense and they're three to six times more atherogenic. So these will infiltrate your arteries at a higher rate and cause earlier and earlier atherosclerosis. These are like the 50-year-olds and 40-year-olds who never smoked, never had heart disease and like, oh my God, what happened? How did I end up with triple bypass, like yeah, that's weird. Or aortic valve replacements it makes your aortic valve tighter and tighter and tighter over time. You know way faster and you don't really have any other reason for it.
Speaker 1:Because I mean, listen, I'm just going to say we talk about, like diet and exercise it's all very important but, like heart disease, number one cause of death, right, like in the country, worldwide, right, like this is the big and it's gotten like we said. It's kind of we're getting, we're understanding and maybe getting better. Back in the day it was just kind of like, oh well, but it's still like the number one.
Speaker 2:This is super important to make sure you keep your cholesterol. It's still the number one killer, but cancer is about to surpass it. Cancer deaths did come down too, but they're kind of creeping back up again. But heart disease deaths are still coming down, but technically, as of now, still the number one killer, but pretty soon it's probably not going to be.
Speaker 3:Well, and of course we're going to see an increase in cancer deaths just because people are living longer and longer.
Speaker 2:Right, and they have to die of something eventually right, right so that's the other thing with like reverse causality. A lot of these people I'm sure you've seen them too they're like well, people with higher LDL live the longest I'm like come on.
Speaker 1:Well, yeah, because cancer like drops your LDL right.
Speaker 2:Like doesn't cancer just like plummet your cholesterol? For example, if somebody comes to me I mentioned this a little earlier they come to me and they get their hdls like 10 and their ldls like 22 and they're not on meds they're not anything like something's wrong.
Speaker 2:They have cancer. They usually have a lymphoma leukemia or cml. Aml, like any of the blood cancers, can cause your hdl and ldl to be super, super low or any other cancer. That's end stage. I say. Say you have liver cancer, lung cancer, brain cancer, whatever really end stage cancer. You're super malnourished, you have no appetite. You just kind of lay there wasting away in bed. You have no muscle, no fat. You're just at death's doorstep. Those people have the lowest LDL cholesterols and these people are like well, people with low LDL die so much they're not dying from the LDL, they're dying from cancer. They just also happen to have very low LDL. In statistics you guys know this, but maybe for the audience that's called reverse causality. A didn't cause B, b caused A. The cancer made the.
Speaker 1:LDL low. Yeah, that's like saying chemo kills you. People who have cancer get chemo, but the chemo isn't causing the. As soon as my mom started chemo.
Speaker 2:she died Exactly.
Speaker 2:She had cancer, she also had a malignant and deadly cancer where 90% of people generally die. I mean, I'm sorry but it's true. And then the other one is like what about this Swedish cohort study where the people who lived to over 100, there was 1,400 of them left and they all had high cholesterol Like, okay, that's survivorship bias? First of all, they didn't check people's cholesterols in that cohort until they were above 65. So you're already self-selecting the ones that are probably going to live the longest, especially since they're born in 1920. At this point. You know, back then, if you made it to 40, that was great, you know. Made it past 50, that's amazing. So that was great, you know. Made it, made it past 50. That's amazing. So the ones that were going to die of heart disease probably already died. Second of all, if you look at the supplemental tables in that Swedish cohort the people who lived to a hundred, their cholesterol was three points higher than the ones who weren't. So they were like 203 versus 200.
Speaker 1:Like come on.
Speaker 2:You really think that that's like a difference? Like you guys are insane. There's no difference between a 203 and a 200 in terms of cholesterol, longevity, living disease free and heart disease free. Like you guys are just insane? You're just looking for stupid stuff. They're like yeah, but it's an aggregate population. Like dude, it's only 1400 people, hundred people, like literally. Of that study you pick, there was seventy thousand people that started. Of the ones that made it to a hundred, there were fourteen hundred of them left and the one out of you measured them. Their cholesterol was just three points different than the ones who died at 95 or 80 or 85. And you know they all made it past 65 because they didn't even check their cholesterol until they were past 65 anyways people.
Speaker 1:They already have like a confirmation bias, they're just looking for something they just look for something that's right, but also the problem is they.
Speaker 2:The problem is that they spread this and it sounds like it's truthful right.
Speaker 1:You know what I mean.
Speaker 2:You get this doctor on there's like a gynecologist. He's like well, look at this, bloody folks, we've got these people that live to 100 and they have the highest cholesterol ever talking about come on, dude, are you like? Why are you first of all talking about cholesterol? You're a gynecologist. You should be talking about vaginal secretions and bacteria in vaginas. You know like? I don't talk about that stuff cause I would be talking out of my butt, but I don't know.
Speaker 1:It's so great. But for the normal people who are like okay, I understand that this is a problem, like what you know give us. We talked about saturated fat. Right, like you reduce, you know, your saturated fat, that's something you can do. You know you can increase your fiber, right, that will, you know, have some effect on cholesterol.
Speaker 2:So there's 11 or 12 different things you can do to lower your cholesterol. Naturally, this is like people are like you know you just push stentons like dude.
Speaker 2:I have an entire series of how to lower your cholesterol naturally. How do you do it naturally? Don't watch it like no one watches the good stuff. They're like you just push stentons. Go watch the other videos, like a thousand of them well, three thousand now, but there. So there's. There's about 11 or 12 ways you can lower your cholesterol naturally. The first one, which has the biggest effect size yes.
Speaker 1:Let's go from top to bottom. What are we doing?
Speaker 2:The biggest effect size is what you mentioned just reduced saturated fat intake. It depends on what diet you're coming from. Obviously, if you're coming from a diet that's very high in saturated fat, you are going to notice the biggest difference in terms of your cholesterol Even up to like 20, 20 to 30% sometimes. If you're like one of these butter chuggers, you're going to notice a significant difference in your cholesterol.
Speaker 1:Right, so we're talking butter like fatty meats, you know, like bacon and stuff.
Speaker 2:Any fat that is solid at room temperature is saturated. It's a super easy way to remember it. Most of them are animal, but there are tricky ones, so you're talking like butter, bacon, cheese, lard, ghee, tallow. The tricky one is coconut oil, because it's not animal.
Speaker 1:I've seen some kind of like mixed stuff on that, but at the end of the day it is still a saturated fat, so you know it's still good.
Speaker 2:There is mixed stuff on whether it's cold pressed and how truly virgin it is and all that, but still the American Heart Association final decision, like guidelines, was like just avoid tropical oils. Palmitic and coconut are basically the two. They are solid at room temperature, they have been shown. So there have been substitution studies where they take butter and they replace it isocalorically with olive oil and or coconut oil. Oh okay, the substitution from butter to olive oil reduced LDL cholesterol the most. From butter to olive oil reduced um ldl cholesterol the most. And the substitution from butter to coconut oil did reduce it, just not as much as going to a poof or moofa right, no mono or polyunsaturated fat, and olive oil would be a little more mono?
Speaker 2:um, there is saturated, obviously, about 10 of it is, but going to like canola oil, which is actually even more mono? Um, well, actually it's more poly. They actually had lower LDL cholesterols. They've done like head-to-heads between olive oil and canola oil and it actually reduced LDL more.
Speaker 1:I know it causes all the inflammation in the body and makes you completely inflamed, which has never been proven.
Speaker 2:They've actually checked inflammatory markers and it has not been proven to be true. They actually did a study for all the seed oil haters. They did a study where people consumed 500 extra calories for just two weeks during the holiday season between Thanksgiving and Christmas.
Speaker 1:I'm sorry, 500 calories is a lot yeah.
Speaker 2:Between Christmas, the 10 days between New Year's and Christmas, they had people consuming an extra 500 calories of pure canola oil, linoleic acid you know the one they hate. Yeah, they actually lost weight and for six months had lower weight than their the the placebo harm. So eating 500 calories more of linoleic acid, for whatever reason, caused more weight loss and or less weight gain during the holiday seasons, but sustained and better weight loss over the next six months too. But back to our how do you lower your cholesterol? So reducing saturated fat is going to be your biggest difference effect size. What have you Other things that help, sort of, like you said, more soluble and unsoluble fibers.
Speaker 2:That helps. Part of it is it kind of binds up the fat and maybe makes you poop it out. Part of it is like plant sterols that are in the fibers, which is another one. Number three would be plant sterols. When you consume plant sterols as actual plants and not supplements, they compete with cholesterol for absorption. Now, as long as you're eating it as an actual plant, if you're taking Benicol and the actual supplements of this stuff, it can be a problem. There are some people who are hyperabsorbers of sterols the plant ones when you take it at such a high dose. It's called cytocysterolemia or phytosterolemia. You actually get really bad atherosclerosis from eating like Benicol and whatnot two, three grams a day of actual plant sterols. So you just want to eat it as plants. That's very helpful. Eating more fruits and vegetables, you know those two actually help. That is probably like number four Less smoking makes a big difference.
Speaker 1:Smoking yeah.
Speaker 2:You know, obviously for various reasons, exercise helps. It doesn't like lower cholesterol a lot. You'd have to exercise a crazy amount to actually lower cholesterol. But, like I said, if we're stacking multiple things, every little bit can help, so adding uh exercise can help. The amount of exercise you'd have to do to actually lower cholesterol is very difficult but exercise is good for a lot of reasons.
Speaker 1:You stack it up, it's good exercise, please exercise for sure, and obviously for longevity.
Speaker 2:You know endurance training and lifting weights both have been shown to improve longevity in no matter how you measure it, whether it's VO2 max, six minute walk test, how long you last on a Bruce protocol on a treadmill, you know all those kinds of things definitely help how fast you can jog a mile, whatever. Or you know 6,800 meters in Rob's case, whatever. Or you know 6,800 meters in Rob's case, you know. So those things matter. Social determinants of health also matter, like do you live in a food desert? Do you have?
Speaker 1:income. Do you have good?
Speaker 2:medical insurance. You know what is your social conditions Genetics? Obviously you can't really change those. There was a couple more. I'm trying to think of them now. Off the top of my head, I think we've been probably 10 or 12.
Speaker 1:Um, but I know, like dietary cholesterol, we've kind of gone away from that being like as much of an issue. Now there are people who, again like you, kind of talked about hyper absorbers with plant sterols or people are hyper absorbers of dietary cholesterol, but in general it doesn't have much of an effect it depends.
Speaker 2:So you know everything is it depends. Sorry, right, if you want to be like honest and not lie to people. Um, so the 2015 guidelines the dietary guidance is the us ones made a mistake in the way that they worded it. They said dietary cholesterol is not a concern for over consumption, so people took it as well.
Speaker 2:That means you can eat all you want, right, they ended they have had to correct that and backtrack but what they meant and they overconsumption so people took it as well that means you can eat all you want. They have had to correct that and backtrack but what they meant and they had to reissue a couple more statements saying listen, we said it's not an issue for overconsumption because people in the United States don't eat that much Like men eat 282 milligrams a day.
Speaker 2:Women were like 272 milligrams a day, there's no concern that we're going to be eating 400 or 300 or whatever. So people ran with it like, oh, you could eat all the dietary cholesterol you want, it's not a concern. But they're like no, it's not a concern for overconsumption, it is a concern in general, just not that much. Now there are been a lot of influencers and people that say oh my God, you can eat all you want.
Speaker 2:Peter Atiyah does that and some other people. You can eat all you want. Peter Attia does that and some other people. Probably not going to be a big deal, but the new guidelines the 2020 to 2025 guidelines have come back and said keep dietary cholesterol to as low as possible. Literally, the exact phrasing was as low as possible. The reason is there's lots of studies out there on dietary cholesterol. Now if you are a hyperabsorber, you're going to absorb more dietary cholesterol, but in general, you may synthesize less. So if you compensate from one end, or like if we put you on a statin, for example, your body will try to absorb a little more. It can't keep up because it's not going to be able to because the absorption mechanism is not that potent. So we generally tell people try to keep it under 300 milligrams a day. In the us and I'm assuming canada is very similar most people probably don't eat over 300 a day. One egg, for example, is 187 milligrams I guess, which is why generally.
Speaker 1:The american heart association recommends like one egg per day which I like to only say that in videos so people can go. I eat 20 eggs a minute. What are you talking about? You know like it's people that opportunity because they flip the fuck out yeah, eddie abu is another one of these bizarre people.
Speaker 2:Um, but yeah, one or two eggs a day, okay, probably not going to make a huge difference. Shrimp, for example. So the problem is like cholesterol is found in weird foods like octopus. You know things we generally don't eat. Yeah, eggs, shrimp 100 grams of shrimp has about 180 as well. Okay, you know 180. 189 milligrams.
Speaker 1:Basically, what I saw in the research was that, like when you go super like, it's kind of like a sigmoidal curve, kind of like saturated fats, where if you have like, if you're like a vegan, a plant-based, where you just know dietary cholesterol, and then you add in dietary cholesterol, it can possibly increase your cholesterol.
Speaker 1:Yeah, it will go up. But if you go from two eggs a day to three eggs or whatever, you know, like you go in these shifts where we kind of normally sit, it's not going to be as big of a difference.
Speaker 2:Right. Definitely depends on what diet you're coming from. That's how it is for most things, right. But if you're coming from a diet that's mostly plant-based with almost no dietary cholesterol and you start adding it, for sure it's going to go up and at least you know transiently and probably maybe over time it'll adjust. If you come from a diet that's a lot of saturated fat to begin with, it's probably not going to make a huge difference if you're already downing butter and ribeyes and you add another couple eggs cheeseburgers.
Speaker 2:You know, putting, putting a poached egg on a cheeseburger probably not going to be the end of the world, but it's still more saturated fat, because there's saturated fat in eggs too you know, and the one thing I will tell people like the white part of an egg is actually pure protein yes, just very yeah, like if you're looking for a food that is a good protein to calorie ratio.
Speaker 2:Let's say you're cutting and you're trying to lose weight. If you want the most protein for your buck without a lot of calories, egg whites is really good. Whey protein you know certain ones depending on what else they add to, it can be fantastic. Faya Greek yogurt is another one of these. It's like almost pure protein.
Speaker 1:I will tell you one of my favorite. Like vegan sources. Oh, you ever tried TVP, textured vegetable protein? It's a little weird and it can be hard to find, but it's it's made from soy and it's like pretty much just protein and fiber, like those are the only two things and you just treat both good.
Speaker 2:Yeah, exactly, they're both great.
Speaker 1:Like I do it, instead of like a ground beef for like uh you know, like tacos or like whatever. You're making that sort of thing like a chili. I'll just add TVP to it. It's a bunch of protein, some fiber.
Speaker 2:How do you get it? Where do you get it from?
Speaker 1:So you usually get it in the baking section. It'll be like it's weird, it's like with like the flours and stuff like that?
Speaker 2:Oh, that is weird. Yeah, it is very weird.
Speaker 1:Look up TVP textured vegetable protein. A lot of people don't know about it. It's like you have to season well because it doesn't taste like really anything. You just kind of rehydrate it.
Speaker 2:It's just got a very bland taste to.
Speaker 2:It just doesn't taste like much and it's very cheap so another thing you brought up which is important is the substitution. So, like some of the say, somebody wants to make chili, do you know that if you use ground turkey, no one's going to know the difference? Or or if you make burgers, if you you mix like, let's say, 20% beef, 80% ground turkey, no one is going to know the difference. Or just buy the 97%, you know, fat free beef, you know. But like it's not that hard to reduce saturated fat intake. If that's your goal, you just have to be like cognizant of it and be like trying to do it Right. Um, so there are lots of substitutions. You can do things like that that really reduce saturated fat. But like, out of all of these things, the single most important thing is reducing saturated fat. The other stuff is all like a one percent here, two percent there, gotcha. None of it is as important as reducing.
Speaker 1:So it's like genetics is the most important which you can't do anything about and then you reduce your, you know, like familial hypercholesterolemia?
Speaker 2:there's not. You just got to be on meds.
Speaker 1:I mean you should do all this stuff anyways.
Speaker 2:But it's malpractice not to be on meds, certain types, the rest of your genetics. If somebody has polygenic, which means like multiple little genetic things here and there, that gives you a cholesterol, let's say like 120 130 it's high, you know's not insanely high. But those people with certain lifestyle modifications cutting fat, exercising, cutting smoking, losing weight, things like that will make a difference. So that's the vast majority of people. I'd say 90% of people have some polygenic baseline cholesterol.
Speaker 1:It seems to me that why do humans have such a high cholesterol? Like it just seems like when you compare it to like other animals, it seems like we just have. I've looked, I've kind of looked in some things on this, like we just kind of have, like what is it when? A, when a baby, when, like your children, like we need the most cholesterol, right, and our cholesterol is like what, like 60 or something, like I don't remember what it is Not even 60 or something like I don't remember what.
Speaker 2:it is not even so, a newborn need. A newborn is, you know, and in utero even they're making brain, right, they're making my, they're making, yes, they make brain good your brain. Your brain is like mostly cholesterol, right, yeah, and the brain.
Speaker 1:Cholesterol has nothing to do with circulating cholesterol, so all these people always say that like you need more cholesterol in your blood, because everything's made of cholesterol and you're like, yeah, every cell can do that, can make its own cholesterol anyway that's another thing.
Speaker 2:Like they, they want to like, say well, doesn't you, don't you, don't you need cholesterol for your brain, like your brain makes its own cholesterol. Yeah, it has nothing to do with circulating cholesterol and the cholesterol in your brain actually has a half-life of five years. A lot of liam's cholesterol in his brain was from infancy, like you know.
Speaker 1:His half life, so you need. So what do babies? What are their cholesterols like when they're born?
Speaker 2:newborns cholesterol is between 20 and 40, and I'm talking 24, what like.
Speaker 1:Why is our cholesterol when we're older so much higher. What?
Speaker 2:because people are like you know. You're saying evolution got it wrong. Well, evolution didn't care for you to live past your reproductive years evolution.
Speaker 1:The point of it is for you to pass on your genes. As soon as you do that, you can drop dead, it doesn't matter yeah, goodbye after the age 40, 45, see you later.
Speaker 2:You know, like the mummies, people are like no, we didn't have heart disease a hundred years ago. Are you like retarded, I'm sorry. Are you like a idiot? Like what do you mean? Like, first of all, nobody lived past 35 or 40 anyways, but they all died of heart disease. Like, literally, that what killed them? Like, if you go, look at the mummies, I have a whole chapter on the mummies in my cholesterol book too. I got to send you guys all these links, because there's like 700 links linked in this book that you could click on and read for yourself. But they did a cohort study on the mummies about 142 of them and they looked at like what did they die of? They did all this fancy imaging of their hearts. They concluded that all of them basically had heart disease. The average age was 42 and they died of heart disease and that was what killed them. So people are like, well, you know, back then, like come on, people, and that's the thing.
Speaker 1:It's like richer people back then, like the pharaohs and stuff. They were the ones able to eat all the meat and, like you know, and their servants did not.
Speaker 2:they ate mostly the and they actually had longer lifespans than the actual gods. They were actually gods. Yeah right, they lived longer than their gods because they did not eat the plants Long enough to get buried with them.
Speaker 3:Some of them, absolutely, and that's how they knew the difference.
Speaker 2:They had longer bone ages and whatever carbon dating or whatever.
Speaker 1:So yeah, just basic, but that's why I call myself the plant slant. You know.
Speaker 2:So, after the blue zones, you know, just like, focus on plant foods, I still eat everything else, all that stuff, but like just, you know, beans and nuts you probably are what's called a flexitarian, where you mostly and I would consider myself that you eat basically mostly plants, fruit, vegetables, fiber, all that kind of stuff, maybe a piece of salmon here and there, maybe a chicken breast here and there. Whatever, I do try to get enough protein, so I do a lot of whey.
Speaker 1:Yeah, I get shrimp or yogurt or whatever. It is Right.
Speaker 2:Fire Greek yogurt, whatever you have to do to get the protein without the stuff that you want. But I just ate some lentil tacos with mushrooms and onions and chickpeas are amazing, oh, like mushrooms and onions, and that's where my chickpeas are amazing. Oh man, chickpeas are so good and a lot of that is Mediterranean cuisine. Like a lot of people are like how do you know? Like my parents came from Syria, that is, on the Mediterranean, and three of my grandparents lived to one oh five, and one of them was one oh six.
Speaker 2:Wow, Really so you know they didn't have exact records back then because it was like a country that third world. I know we don't say that anymore, but it was a third world country and they didn't have perfect records. But we know when our grandpa was 97 for sure, and then he lived at least another six or seven years.
Speaker 1:We don't know exactly when he was born, but we know he's definitely well we might not have his apo b, but we know he was doing pretty well.
Speaker 2:The funny thing is, like the other grandpa, so three of them lived to over 100. Our other grandpa was really bad diabetic smoker, you know, didn't really take care of himself. Died at 51 of a massive heart attack. So you know that kind of stuff makes a difference. You know genetics plus stuff you do.
Speaker 1:Right, exactly I always heard the phrase like genetics loads the gun while your environment fires it.
Speaker 2:so like don't, you can try not fire the gun right see, okay, rob, in america everything is about guns, so we have to bring it all back to that, just in case you were confused over there, what was going on? It all has to be about firearms anyway loads the gun and then your environment pulls a trigger or something like that yeah, exactly the other thing that I think a lot of people they don't want to accept that a lot of this is genetically determined, for whatever reason why, don't you just tell people to eat right?
Speaker 2:yeah, who tells people to eat wrong? Like am I suggesting for you to eat like poop, or donuts supplements like I don't sell testicle supplements but I'm telling you to eat right and that you don't need the testicle supplements. But like where do you get that I'm telling you you have to eat wrong? Like I don't, I'm not.
Speaker 1:But it's because we say because earlier you said it's okay to eat a pop tart, which means you are wrong, and you told people you should only eat Pop-Tarts. That's how it works.
Speaker 2:Yeah, and when I posted this video, I posted a video explaining Dr Mark Haub's Twinkie diet. Literally, the head of nutrition at Kansas State University wanted to prove a point and ate only Twinkies.
Speaker 1:Twinkies, Oreos, Doritos. He took a protein mix or something like that.
Speaker 2:He ate a high drink 100 grams of protein a day to not lose muscle. Because he's the head of nutrition, he's not an idiot.
Speaker 1:He ate only Doritos, Twinkies, Oreos. He lost 27 pounds, I believe 28 pounds in 10 weeks.
Speaker 2:28 pounds, yeah, and they're like you're an idiot, you don't know what you're talking about. Like, stop, I'm reporting on a study, I'm not telling you. This is optimal.
Speaker 1:And his cholesterol improved, Like his LDL went down. I'm pretty sure.
Speaker 2:All of his markers improved with the weight loss. So I'm telling you and this lady went after me, she's like you're just an idiot, you don't know anything, I'm like lady this is not me.
Speaker 1:It's because when you say that, people hear you telling them that they should only eat Twinkies and Oreos and it's like that's not what we're saying. We're trying to prove a point here.
Speaker 2:I'm reporting a study to you that was done by the head of nutrition at Kansas State University, so I tagged him on Twitter. I was like Dr Mark Haub did this You're welcome to argue with him and I tagged him on there, she just shut up.
Speaker 1:oh, that's funny.
Speaker 2:I'm not saying this is optimal. It's like these people. They're like well, you can eat keto and lose weight. Sure, of course it's optimal. Like well, you don't need carbohydrates. Name one nutrient that is essential, it's not carbs. I'm like oh, I know that.
Speaker 3:Yes, not telling you, you have to do that but there's so many times you get the comments that are. You always tell us that everything's good for us. Why don't you tell us something that's bad for us? I?
Speaker 1:know Overconsumption of anything that's bad for you. Don't do that.
Speaker 3:We literally don't tell you that something's good for you. We tell you that you can eat it in moderation.
Speaker 1:You're saying aspartame is good for me, you idiot. What are you talking about? I said it's not harmful.
Speaker 2:They think you mean you should order a truckload of aspartame and try to consume all right in 24 hours, but it's like bizarre they have. But the problem is you know why? I think part of it is all these other influencers are so extreme, like bobby fave is like don't ever eat this and then like 10 minutes later, like don't ever eat this. It's like they want us to be like don't ever eat this, like wait why yeah, everybody can only think extremes now.
Speaker 3:But if you have rules, people have to follow them, like.
Speaker 1:That's the thing, that's how you get followers. Is you got to have rules?
Speaker 2:Rob and I were talking a little bit off air before you jumped on. I think the extreme stuff does so well on social media because it's insane, like if I go online and I say you need to inject your own urine into your eyeballs that video will get a million hits. You your own urine into your eyeballs. That video will get a million hits. You'll get billions of hits and views and whatever. But if I go online, be like listen, you just gotta eat in moderation.
Speaker 1:Exercise, lift some weights.
Speaker 2:Ride a bike, do something normal. You know the video is gonna get like two follows right, because the normal information is boring.
Speaker 1:Man, we need that. We've talked so much on so many episodes. We have so many ideas for how to grift people. When we eventually give all this up, we've come up with tons of stuff that we're going to start selling. That's just.
Speaker 2:You could sell urine eyeball injectors.
Speaker 1:We already have what. What do we have? Rob Dragon water?
Speaker 2:One of my patients asked me today one of my patients today was like well, you know what, doc, why don't you sell heart supplements? I was like, first of all, what would I possibly sell that is not illegal to give you over the counter supplements that would help your heart? Give me an idea. He's like well, you know, like statins. Like statins, I know.
Speaker 1:Right, just sell statins, because, as you said I've heard you say many times if a supplement works, they take it and they turn it into a medication and they regulate it and they test.
Speaker 2:So, like your audience might not know, but literally every cardiac medication started out as something in nature Like you're anti-natri because you can't pit and make money off it Like hold on hold on. Let me tell you the most prescribed. It's not even a prescription anymore, but the most used medication in cardiology is oil of wintergreen. They're like no, you can't make money off it. I was like stop, it's called aspirin.
Speaker 1:It's salicylic acid.
Speaker 2:It's salicylate, that's aspirin, it's oil of wintergreen, it's from the bark of a willow tree. People are getting stomach aches and headaches. They'd go like chew on a stick. Sit in a corner and they got better because they absorb it through their mouth, right. They're like well, okay, fine, you know. And then I'm like well, how about lisinopril? Do you know where lisinopril came from? It's like the most prescribed blood pressure medication. It's super potent. It's amazing. It's the viper venom. There's two like no, there's hundreds. Yeah, like statin medications like you guys are just statin pushers. Like no, first of all, statin is yeast. It literally came from mold, just like penicillin. They're like really well, you know why you guys pen it like so you don't die when you go like yeast, like part of yeast. Like the problem with red yeast rice is, you know, there's the part of it that is lovastatin, but the other part of it can kill your kidneys, like you know. Do you really want to be taking an unregulated?
Speaker 1:substance, but natural good, artificial bad, dr Aloe, natural good, artificial bad.
Speaker 2:That's basically where we've gotten to in social media All these new injectable medications that cause weight loss and help diabetes. They come from the saliva of a lizard called the Gila monster in Arizona. Like, oh, give me Ozempic, give me Monjaro? Like okay, but you're, you're asking me for saliva of a lizard. Like what do you guys not understand? Like? There's so many other examples. I go through these in some of my videos, but all of these come from nature. It's like well, you, you don't sell cayenne pepper because you can't patent it. I'm like, dude, I would patent it and it would be a medication and we would make billions off of it. They just don't get it. They think it's a conspiracy to keep them unhealthy and harm them and keep these big secrets from them.
Speaker 2:They're like well, other cardiologists disagree with you. I'm like name one, go ahead.
Speaker 3:I always find it hilarious because as soon as you look at any of these socialized medicine countries that whole doctors are in it for the money and they're out to poison you and stuff, it just falls apart.
Speaker 1:You don't get that here you know dr ids gets that all the time like you're just social, like dr it's in fucking england, like they have social meds like.
Speaker 2:What is? They make 20 bucks a day.
Speaker 1:What the fuck what are you talking about? Meanwhile dr ken berry here is fucking telling everybody like, oh, if you have cholesterol you need to go on keto. And I'm just like holy shit.
Speaker 2:Literally. He posted a video in an airport on his way to a keto conference. He's like Johnny here, just had two stents put in his heart. I'm sorry, I shouldn't do that, but he's like Johnny here, just got two stents put. Well, it was kind of funny, johnny here, got two stints put in his heart and the best thing you can do, johnny, is start eating some butter and bacon and cholesterol. Like dude, what you just killed Johnny. Like Johnny is dead. Like, what are you telling Johnny? Like, why are you telling him this?
Speaker 1:There's no data to support what you're saying other than like maybe you're saying you're crazy, but he's the guy that sells the supplements.
Speaker 2:He's the guy that sells the supplements. He's the guy that sells niacin and all those other things that people say. You'll notice most of these carnivore crazy docs. I'm not just picking on the carnivores, there's lots of them. They have. Their medical licenses were all suspended because they're harming patients on purpose. You can't just get your medical license taken away for killing somebody by accident. It happens right.
Speaker 1:But if you are doing it on purpose, multiple times after multiple warnings.
Speaker 2:Then you get suspended. Like literally, it takes a lot for your medical license, right, like as a doctor, to get revoked. Yeah, it's impossible, like, unless you are literally insane trying to kill people, like injecting them with your urine your own urine or hiv, or something insane like that. You know, I mean it's, it's nuts, but then you're like.
Speaker 1:Well, he said it's okay to eat butter well, he's a doctor and he said it Like holy shit, no but the best is like, oh, other cardiologists disagree with that.
Speaker 2:I'm like, okay, name one. And then they never respond. I'm like just name one, Just pick me one single cardiologist that does not think that cholesterol causes heart disease, and then the only one they can come up with is Asim Mahotra. I'm like they kicked him out of England because he's insane. He has psychological issues. Did you hear his interview on Rogan? He's nuts.
Speaker 1:Oh, was he on Rogan? Of course he was, of course he was.
Speaker 2:Rogan is like he gets the craziest of the crazy. You know, like normal cardiologists would not.
Speaker 1:It's not clickbait, because again it's not clickbait, you know, because again it's boring it's boring, like people are just real.
Speaker 2:Medicine is like if you follow me around for a day, it's not that exciting. I mean, it might be for some people because you get to see cool stuff, but it's not that exciting.
Speaker 3:I mean, I'd be all for that I haven't seen it for a day.
Speaker 1:But like I get your point, like if you're like, oh, this is it, like it's just met, you know I mean come on down, I'll be more than happy to take you around.
Speaker 2:But it's like we tell people to eat right, lose weight, try diet and exercise. If it's not working for you, we have other solutions. We can force you to lose weight. Now, like literally, I can make you lose weight pretty much effortlessly with the kinds of medications and therapeutics we have. Now you don't even need gastric bypass anymore. You literally just can sit at home on your butt and lose weight without even trying, and I can force you to do it. It's not even hard anymore.
Speaker 3:But it's definitely you making money from the big pharma, and not these other doctors that are selling supplements.
Speaker 2:I know. No no, and that's the funny thing is don't you think big supplement pays these idiots?
Speaker 3:Like they are big supplements. I love people that If you're making a hundred, million dollars selling testicle weed or whatever Testicle weed.
Speaker 1:I love testicle weed.
Speaker 2:Isn't that money?
Speaker 3:No, I love the people that bring up David Sinclair and nicotinamide mononucleotide and there was like well, this guy is like he's a specialist in genetics and longevity and stuff and he tells you to take NMN. Who do you think owns the patent for NMN? David Sinclair's company.
Speaker 2:And he sells tons of stuff. But you know who you should get on the show. James Brenner is an actual longevity, real expert. He goes after Sinclair all the time. He's like nothing this guy says is real. He's like. I've asked to debate him a million times, he just keeps blocking me on every point. But James Brenner, he was on Simon Hill. You know Simon Hill the Proof.
Speaker 1:Yeah.
Speaker 2:Yeah, he was on that show and literally, if you watch that episode or listen, to it.
Speaker 1:It's a podcast too.
Speaker 2:He goes after Sinclair. He's like listen, this guy's just full of. He's using one cell organisms and extrapolating it to humans, like not even rats At least some people are doing like mammal studies, like on rats and rabbits. This guy's like in a totally different universe with his bizarreness. But yeah, he's like selling all this stuff New York Times bestseller, all that. That's like I think people need to be careful with the money. Like big supplement, big cattle, big agriculture, big, big, big, big, big, whatever. But then, like you got this poor little family doctor. That's like you know I'm trying to help you but you want to believe in some weird conspiracy theory, thinking that I'm getting paid to like pretend to help you but really try to kill you, which is that's why they always say, like, follow the money.
Speaker 1:It's like it's money with seed oils or statins or whatever. And then you're just like, but what about? Like the supplement industry? Like that's just as bad if the industry is almost a trillion dollars.
Speaker 2:I mean, the last s my saw was like 750 billion or something like that. Um, it's sad that people buy into this stuff.
Speaker 3:Yeah, and like since claire he, um, he has that uh infamous thing where he is. Uh, he did all this genetic or biotesting and his biological age was like 20 something and, great you think, owns the company that did that biological testing not his yeah so I'll tell you guys a funny and crazy story.
Speaker 2:One of my friends sells supplements, right, and he did this all throughout college. When Amazon kind of first started, he would send the label to China. They would label these sugar pill bottles and send him 5,000 of them every month or so, you know, every month or so. First he labeled them like testosterone booster. You know didn't work. A lot of sales. Then it was like B12, you know, b complex something didn't work. Then they finally picked some gimmicky thing that actually worked and he sold out. So he just kept doing that. Just buy the same pills.
Speaker 2:We'd send him a different label each time, call him something and people would buy it. The problem is people would leave reviews and say, like this worked. The placebo effect is super potent. A lot of people I'm sure you guys get these people all the time like, well, I did clamp pepper and it worked. Like, first of all, what do you mean by it worked? Second of all, even if you know you're getting the placebo, there have been studies done where people know they are receiving the placebo and they still reported better outcomes.
Speaker 1:They say they felt better they went away, or whatever it might be. Whenever you take a supplement for something that already already means you're like I want something to work, so maybe I'm going to do other things as well. I'm going to take the supplement, but I'm also going to eat a little bit more healthy, I'm going to exercise more, and so all these things go together and then you can't say, just it's because of that, like, oh, it drives me crazy, but then they'll look at a medication and find some obscure side effect that like 0.001, yeah, and they're like, oh, but I don't want to get that.
Speaker 2:Or like the best. I had a patient one time this is funny, you guys will love this this patient was like you know, 400, some odd pounds. I'm like, hey, uh, you know, you know, we've got your cholesterol down. Your diabetes is sort of under control, not really. Do you want to try one of these new weight loss meds? And he looks at me and he goes. I heard they have a lot of side effects.
Speaker 2:And I looked at him and I was like, okay, well, being 450 pounds also has a lot of side effects. So, like pick, do you want to stay 450 pounds and possibly die of need you know, need knee replacements and joint replacements and heart attacks, strokes, blood pressure, cholesterol, diabetes, all that stuff? Or do you want to take these medications that, like, less than 100% of people get anything weird from it and possibly lose 350 pounds or whatever it might be?
Speaker 1:So I don't know.
Speaker 2:Sometimes they just watch too much Jillian Michaels.
Speaker 1:Jillian Michaels.
Speaker 3:When I was looking into finasteride, one of the side effects is erectile dysfunction and there was a study on it where they had a group that they actually told erectile dysfunction was one of the side effects and another group that they didn't tell anything. And the group that had been told had more erectile dysfunction the group that didn't get told had like no erectile dysfunction that's called the druscebo effect.
Speaker 2:Like you know, you're on the drug, you get the drugcebo druscebo effect. Like people who are on statins think they are going to get myalgias or muscle aches because they think statins give you muscle aches, so they report it.
Speaker 2:But a lot of times when they do the studies they just tell them they're on the statins when they're really not on it and similarly they report the side effects. But it's just weird that like people are willing to risk their lives not losing weight taking weird supplements that might not contain like turmeric, for example and I'm sure you guys you guys had the supplement lady on not that long ago but there have been studies on turmeric that show that most turmeric it comes from india and it's very contaminated. It actually includes like liver toxins that could cause liver failure, but people are downing it as if it's harmless. People think supplements are completely harmless.
Speaker 1:Yeah, that's true.
Speaker 2:My friend from medical school was the hydroxycut spokesperson. Rob might remember hydroxycut.
Speaker 1:I remember hydroxycut.
Speaker 2:Yeah, it was this guy, super muscular hockey player, but he's a doctor now. He was a spokesperson for it. He was on the commercials on TV. Literally it was like ephedra, and it was back when ephedra was legal or technically legal, but it was ephedra and something else.
Speaker 1:Technically legal is my favorite kind of legal.
Speaker 2:Yeah, three people died of liver failure because people thought you could just take as much of it as you want, because it's natural and it's a supplement. They took so much of it that they killed their livers and he stopped being their spokesperson and was like this is insane. It's no longer allowed to contain that. Obviously, now it's just like a bunch of caffeine and yerba mate extract or something.
Speaker 1:Oh, but we found new things that work deep in the Amazon rainforest. We found this one, Ruth.
Speaker 2:The best is when Paul Sal saladino went to germany supposedly secret library and he translated it into english and there was this one little paragraph about liverweed testicle nonsense and he made it and it works like dude, are you like serious?
Speaker 1:the german book is like watch this and take this seriously. Because it's entertaining.
Speaker 2:What is the weirdest supplement you guys have ever heard of? I'll tell you mine. I've got people with like lists that are like 30 pages long the worst one, most bizarre. There's a bunch, but one of them was horse hoof shavings. One of my patients was taking horse hoof shavings. One of my patients was taking horse hoof shavings. Like literally when they cut off part of the horse's hooves, they put that in a plastic capsule and they eat it Like they've been walking in manure. You like you don't understand that, but that was one of their supplements. And there's all these weird, bizarre weeds.
Speaker 1:And my favorite, my favorite type, I would say, is would say is the people that give themselves a tapeworm and it comes in supplement form. So you don't lose weight. The tapeworm just eats the food. Because you're like look, I can eat as much as I want and I don't gain weight. And they're like, yeah, boy, you also have a tapeworm inside your body.
Speaker 2:Heaven forbid you take some Ozempic Jeez.
Speaker 1:You'd rather take a life-threatening worm instead of? Oh, you're just being a naysayer, Dr Aloe.
Speaker 2:You just don't want people to be able to eat as much as they want, lose weight. I'm just a pill pusher. I'm a pill pusher.
Speaker 1:Pill pusher, you don't want these tapeworm supplements.
Speaker 2:I just don't want people to get healthy right.
Speaker 1:Because that's the other thing. I don't want people to get healthy, because that would be less money in my pocket, of course. That's the reason. Meanwhile, I'm going to sell these tapeworms and make a fortune.
Speaker 2:A cured patient is a business lost, or whatever they say. Like right, that's exactly what.
Speaker 1:I'm trying to do.
Speaker 2:So I can keep making money off your freaking $2. Like what are you?
Speaker 1:people saying so good, love it, love it. No, these people are nuts, but.
Speaker 2:I don't know how to combat this other than just keep making good content. That's reasonable.
Speaker 1:Yep, and that's what we're trying to do right here is basically just give out as much decent information as possible. If people wanted more decent information, where could they find you, dr Allo? For more stuff, just search.
Speaker 2:Dr Allo anywhere.
Speaker 1:I feel pretty good about that. One Go ahead.
Speaker 2:Search Dr Aloe anywhere. I feel pretty good about that. One Go ahead, just search Dr Aloe anywhere. D-r-a-l-o.
Speaker 1:A-L-O.
Speaker 2:D-R-A-L-O, dr Aloe Like the lotion, but no E at the end.
Speaker 1:Like the lotion. You're like Dr Aloe, like the lotion.
Speaker 2:No, e I literally tell patients that that's your stripper name. Because I say to patients I'm like, yeah, they're like what's your name? Again, I'm like Dr Aloe. They're like Dr Hallow Hallow. I'm like no, like the lotion, but there's no E on the end. They're like, oh, dr Aloe.
Speaker 1:And then they get it and it clicks Like.
Speaker 2:Aloe Vera, but want somebody that knows absolutely nothing and is a conspiracy theorist, is trying to kill you to make more money off of you. Please subscribe oh goodness. That's what I'm called every day.
Speaker 1:I know.
Speaker 3:And for people who want to see these cholesterol have sex. What's your OnlyFans? What's your OnlyFans? Oh gosh, cholesterol have sex what's your only fans?
Speaker 2:what's your oh gosh? I don't know that I've explored that option, but I'm not. I'm not gonna sell supplements ever it's at sexesterol there is such a thing oh goodness, the lipoprotein molecules are having sex with one another for sure so good, this has been a fun episode. I've been on a lot of podcasts, probably the funnest ones so far.
Speaker 1:I just can't have fun with it. A lot of them are way more serious. No, serious is boring. I listened to your one on the proof with Simon Hill. That was good. That was good yeah he's a good dude.
Speaker 2:I like Simon. He's like. I found you on tiktok, oh yeah, yeah, I kind of found you on tiktok too. What is a cardiologist doing on tiktok? It's like you know what. I really have no idea.
Speaker 1:Somebody told me to get on tiktok losing my mind, losing my sanity, is the answer you should have given him.
Speaker 2:No, no, I'm trying to kill people to make more money off of them, that's literally what I'm doing with my, my evil statins no, because, because if I keep you sick, then I make more money.
Speaker 1:Oh, that's another good one. Yeah, it's like they keep Big.
Speaker 2:Pharma keeps you sick to make money off you. I'm a spokesperson for Big Pharma where clearly you could go online and look that up. It's literally like go to openpaymentscmsgov. Type my name in. You'll find I've made all of $2. Type my name in You'll find I've made all of $2 from.
Speaker 1:Big Pharma.
Speaker 3:Well, that's just because you don't have a promo code for your statins yet.
Speaker 2:Or I'm not selling, like you know.
Speaker 1:Sell your red yeast rice, man. People always ask about that. I see people ask about either that all the time. Just sell it to them.
Speaker 2:Make some money, Just make my own. Listen all I'm saying is.
Speaker 1:you sell the red yeast rice wherever the fuck it is, and then you say, look, I'm trying to do something. That's natural as well, see, and then people will also like you more and you'll make more money, that's all I'm saying no, then you'll get the he sold out.
Speaker 2:He's a supplement salesman. Now I told you he was. We can sell supplements. Then you're going to get the other haters that are like you're just a sellout.
Speaker 1:Yeah, no, you pretty much. People just hate you Like, whatever you do on social media, it doesn't matter what side you're on I mean you know Paul Saladino and all of them they get plenty of hate, deservedly so.
Speaker 2:Another example of this, which is even more bizarre Lane Norton was on a. He wrote a forward for my book, by the way. Oh, lane norton was on a. He wrote a forward for my book, by the way. Oh he was. He was on a podcast explaining how to lose weight. Literally some super benign thing his nails were painted because his daughter likes to paint his nails for him before his competitions. Literally no one asked about weight loss. No one in the comments said anything about the actual science and data and studies. They were obsessed with why he painted his name yeah, I know like are you people like idiots, like why is this even a concern of yours?
Speaker 2:I can dress and do my hair and my beard and my eyeballs and eyelashes and whatever however I want. It's none of your business. If you have something to say about my comments or studies or data, feel free.
Speaker 1:No, I mean, we can talk all day I mean, even like you know you do, I do a video about, like you know, your bobbies of the world or whatever, like I'm like the stuff they're saying is just nonsense. But a lot half the comments are just like, yeah, and he's a gay boy. And I'm like that's not really where I was going with this. I don't care. Even if he was gay which I don't think is I don't care. Why are we obsessed with like this other shit? Oh, his genes are stupid.
Speaker 2:Okay, like I don't yeah, like, and then or somebody would be like. So, for example, I even get offended on behalf of ken berry, believe it or not. They're like well, look at him, he's obese. And they'll find like some obscure picture somewhere and post a picture of him being obese like okay, that has nothing. I don't care, just said, or what he just said, he's also telling people he's not metabolically healthy.
Speaker 2:Why don't you tell him you don't look healthy either? But people, what are you even arguing here and why like? How does this help?
Speaker 1:I don't know it's, it's, it's, it's just arguing that you don't look pretty Apparently, I don't?
Speaker 3:It's all about looks.
Speaker 2:I don't like my like hair sticking up, Like why do you care? I can do my hair However I want. I can dress however I want. I can not dress however I want, Like what is the problem here? But I mean that's why I the soul patch is just rage bait for people to get mad and comment which I'm out now. I'm like insecure, Like eyebrows or something Like what how is this even?
Speaker 2:like an issue, Like I've had multiple people say it now I'm like, okay, did I like get fake eyebrows and paste them on? Or something Like what, what we like? What are they seeing that I'm not.
Speaker 1:It's weird, it's I don't know but,
